von Dr. Haitao Wang
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| [1.] Haw/Fragment 047 15 - Diskussion Zuletzt bearbeitet: 2014-10-12 17:14:14 Schumann | Fragment, Gesichtet, Haw, KomplettPlagiat, SMWFragment, Schutzlevel sysop, Van Oostrom et al 2008 |
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| Untersuchte Arbeit: Seite: 47, Zeilen: 15-23 |
Quelle: Van Oostrom et al 2008 Seite(n): 1380, 1381, Zeilen: 1380: r. Spalte: 16 ff.; 1381: 1 ff. |
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| Interestingly, Pipp et al.[44], demonstrated the importance of FSS in arteriogenesis by means of a porcine ischemic hindlimb model with extremely high levels of collateral flow and FSS. Normally, during the later phases of arteriogenesis, FSS decreases as the collateral diameter increases so that FSS normalizes. This drop in FSS acts as a signal to arrest proliferation and as a result, prevents further collateral growth before an optimal adaptation is reached. Pipp and colleagues [44] demonstrated that sustained, elevated FSS in their arteriovenous shunt model further significantly increased the size of collaterals, thus establishing that FSS is a dominant morphogenic factor in collateral growth.
44. Pipp, F., et al., Elevated fluid shear stress enhances postocclusive collateral artery growth and gene expression in the pig hindlimb. Arterioscler Thromb Vasc Biol, 2004. 24(9): p. 1664-8. |
Interestingly, Pipp et al. [14] demonstrated the importance of FSS in arteriogenesis by means of a porcine ischemic hindlimb model with extremely high levels of collateral flow and FSS. Normally, during the later phases of arteriogenesis, FSS decreases as the collateral diameter increases so that FSS normalizes. This drop in FSS acts as a signal to arrest proliferation and as a result, prevents further collateral growth before an optimal adaptation
[Seite 1381] is reached. Pipp and colleagues [14] demonstrated that sustained, elevated FSS in their arteriovenous shunt model further, significantly increased the size of collaterals, thus establishing that FSS is a dominant morphogenic power in collateral growth. 14. Pipp, F., Boehm, S., Cai, W. J., Adili, F., Ziegler, B., Karanovic, G., Ritter, R., Balzer, J., Scheler, C., Schaper, W., Schmitz-Rixen, T. (2004) Elevated fluid shear stress enhances postocclusive collateral artery growth and gene expression in the pig hind limb. Arterioscler. Thromb. Vasc. Biol. 24, 1664–1668. |
Ein Verweis auf die Quelle fehlt. Ein Teil des Fragments wird hier zum zweiten Mal verwendet: Fragment 006 21. |
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Letzte Bearbeitung dieser Seite: durch Benutzer:Schumann, Zeitstempel: 20141012171439