von Dr. Haitao Wang
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| [1.] Haw/Fragment 010 07 - Diskussion Zuletzt bearbeitet: 2014-10-13 10:57:49 Hindemith | BauernOpfer, Fragment, Gesichtet, Haw, SMWFragment, Schaper and Scholz 2003, Schutzlevel sysop |
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Quelle: Schaper and Scholz 2003 Seite(n): 1148, Zeilen: r. Spalte: 36 ff. |
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| 1.3.5. Remodeling
After the acute phase of arteriogenesis that is dominated by the inflammatory events, remodelling begins (phase 2 of arteriogenesis), i.e., the much slower consolidation of the arterial structure after the final diameter was almost reached. A new elastic lamina is synthesized by the SMCs, and the rebuilding of the media and the formation of an intima begins with the downregulation of the tissue inhibitor of matrixmetalloproteinases (TIMP and MMP)[65]. 65. Cai, W., et al., Altered balance between extracellular proteolysis and antiproteolysis is associated with adaptive coronary arteriogenesis. J Mol Cell Cardiol, 2000. 32(6): p. 997-1011 |
Remodelling
After the acute phase of arteriogenesis that is dominated by the inflammatory events, remodelling begins (phase 2 of arteriogenesis), ie, the much slower consolidation of the arterial structure after the final diameter was almost reached. A new elastic lamina is synthesized by the SMCs, and the rebuilding of the media and the formation of an intima begins with the downregulation of the tissue inhibitor of matrixmetalloproteinases (TIMP and MMP).80,81 80. Cai WJ, Vosschulte R, Koltai S, Kostin S, Schaper W, Schaper J. Extracellular proteolysis is involved in coronary collateral vessel development in dog. J Mol Cell Cardiol. 1997;29:A128. 81. Cai WJ, Vosschulte R, Afsah-Hedrij A, Koltai S, Koscic E, Scholz D, Kostin S, Schaper W, Schaper J. Altered balance between extracellular proteolysis and antiproteolysis is associated with adaptive coronary arteriogenesis. J Mol Cell Cardiol. 2000;32:997–1011. |
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Letzte Bearbeitung dieser Seite: durch Benutzer:Schumann, Zeitstempel: 20141012165805