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MEHR ERFAHREN

VroniPlag Wiki


Typus
BauernOpfer
Bearbeiter
Graf Isolan
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Untersuchte Arbeit:
Seite: 169, Zeilen: 1-2
Quelle: Krueger 2001
Seite(n): 106, Zeilen: 30-35
[Allerdings wird für den spontanen und den induzierten Zelltod von PBL in vivo und in vitro generell die Induktion] der Apoptose verantwortlich gemacht (HASLETT et al. 1985, PAYNE et al. 1994, GASMI et al. 1996, WALZOG et al. 1997, HANNAH et al. 1998).

GASMI L , MCLENNAN AG & EDWARDS SW. (1996) :
The diadenosine polyphosphates Ap3A and Ap4A and adenosine triphosphate interact with granulocyte-macrophage colony-stimulating factor to delay neutrophil apoptosis: implications for neutrophil: platelet interactions during inflammation.
Blood ,87, 3442-3449

HANNAH S, NADRA I, DRANSFIELD I, PRYDE JG, ROSSI AG & HASLET C. (1998) :
Constitutive neutrophil apoptosis in culture is modulated by cell density independently of beta2 integrin-mediated adhesion.
FEBS Lett. ,421, 141-146

HASLETT C, GUTHRIE LA, KOPANIAK RB JOHNSTON & HENSON PM. (1985) :
Modulation of multiple neutrophil functions by preparative methods or trace concentrations of bacterial lipopolysaccharide.
Am. J. Pathol., 119, 101-110

PAYNE CM, GLASSER L, TISCHLER ME, WYCKOFF D, CROMEY D, FIEDERLEIN E & BOHNERT O. (1994) :
Programmed cell death of the normal human neutrophil: an in vitro model of senescence.
Microsc. Res. Tech. ,28, 327-344

WALZOG B, JEBLONSKI F, ZAKRZEWICZ A & GAEHTGENS P. (1997) :
Beta2 integrins. (CD11/CD18) : promote apoptosis of human neutrophils.
FASEB J. ,11, 1177-1186

Allerdings wird für den spontanen und den induzierten Zelltod polymorphkerniger Granulozyten in vivo und in vitro generell die Induktion der Apoptose verantwortlich gemacht (HASLETT et al. 1985, PAYNE et al. 1994, GASMI et al. 1996, WALZOG et al. 1997, HANNAH et al. 1998), obgleich über die einzelnen, regulierenden Kontrollmechanismen immer noch wenig bekannt ist (WALZOG et al. 1997, TSUCHIDA et al. 1995).

GASMI, L., A.G. MCLENNAN & S.W. EDWARDS (1996)
The diadenosine polyphosphates Ap3A and Ap4A and adenosine triphosphate interact with granulocyte-macrophage colony-stimulating factor to delay neutrophil apoptosis: implications for neutrophil: platelet interactions during inflammation.
Blood 87, 3442-3449

HANNAH, S., I. NADRA, I. DRANSFIELD, J.G. PRYDE, A.G. ROSSI & C. HASLETT (1998)
Constitutive neutrophil apoptosis in culture is modulated by cell density independently of beta2 integrin-mediated adhesion.
FEBS Lett. 421, 141-146

HASLETT, C., L.A. GUTHRIE, M.M.KOPANIAK, R.B. JOHNSTON & P.M. HENSON (1985)
Modulation of multiple neutrophil functions by preparative methods or trace concentrations of bacterial lipopolysaccharide.
Am. J. Pathol. 119, 101-110

PAYNE, C.M., L. GLASSER, M.E. TISCHLER, D. WYCKOFF, D. CROMEY, R. FIEDERLEIN & O. BOHNERT (1994)
Programmed cell death of the normal human neutrophil: an in vitro model of senescence.
Microsc. Res. Tech. 28, 327-344

TSUCHIDA, H., Y. TAKEDA, H. TAKEI, H. SHINZAWA, T. TAKAHASHI & F. SENDO (1995)
In vivo regulation of rat neutrophil apoptosis occuring spontaneously or induced with TNF-α or Cycloheximide.
J. Immunol. 154, 2403-2412

WALZOG, B., F. JEBLONSKI, A. ZAKRZEWICZ P. GAEHTGENS (1997)
Beta2 integrins (CD11/CD18) promote apoptosis of human neutrophils.
FASEB J. 11, 1177-1186

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Sichter
(Graf Isolan), Hindemith